DSIP vs Selank

Sleep-associated nonapeptide vs Anxiolytic heptapeptide · Head-to-head comparison

Both are sold for sleep or calm and both come out of the same Soviet-era and Swiss research traditions. DSIP is the more extreme case: fifty years on, nobody established what it acts through.

Side by side

 DSIPSelank
ClassSleep-associated nonapeptideAnxiolytic heptapeptide
StatusResearch compoundApproved in Russia (intranasal); research compound elsewhere
RouteSubcutaneous injectionIntranasal (primary) or subcutaneous
Typical vials2 mg, 5 mg5 mg, 10 mg
Dose range100 mcg to 500 mcg250 mcg to 750 mcg
FrequencyNightly, before bedDaily, as needed
Half-lifeNo human dataNo human data
Dosing unitmicrogramsmicrograms

Pharmacokinetics

No curves are shown, on purpose.

No published human pharmacokinetics for DSIP or Selank. Drawing one curve against an absent one would imply a comparison that cannot be made.

What the evidence supports

These grade how well each outcome has been studied in humans, never how well either compound works. A strong grade can sit on a negative result.

The strongest evidence for any claimed outcome is Human RCT for Selank and Human, observational for DSIP. That gap is the most important line on this page.

DSIP

OutcomeEvidence
Chronic painHuman, observational
Sleep onset in insomniaHuman, observational
Modern replication of any 1980s resultNo data

Selank

OutcomeEvidence
Generalized anxiety disorderHuman RCT
Cognitive and attention measuresHuman, observational
Independent replication outside Russian literatureNo data
Human RCTAt least one randomized controlled trial in people reports this outcome. The result may still have been negative.
Human, observationalReported in people without randomization: open-label, single-arm, retrospective, or case series.
No dataNo published evidence at any level that we could find.

How to choose

DSIP's mechanism was never elucidated

Despite the name, it is not a straightforward sedative, no receptor or pathway was ever pinned down, and research largely stopped in the 1990s.

Selank at least has a proposed mechanism and recent trials

GABAergic and monoamine systems, with Russian clinical work.

Neither has evidence a reader outside Russia can easily check

For DSIP the literature is old and thin; for Selank it is Russian-language and not replicated.

At the syringe

At the default setup on each compound's own page, a 5 mg vial in 2 mL puts 25 mcg on every insulin unit, while a 5 mg vial in 2 mL puts 25 mcg on every unit. That is the number that decides whether a dose lands on a readable mark, and it is set by how much water goes in, not by the compound.

Full references