BPC-157 vs TB-500

Body-protection pentadecapeptide vs Thymosin Beta-4 active fragment · Head-to-head comparison

The most common pairing in the repair category, usually sold and dosed together. Both are animal-and-mechanism compounds with no human trials, and the TB-500 side carries a specific confusion worth knowing.

Side by side

 BPC-157TB-500
ClassBody-protection pentadecapeptideThymosin Beta-4 active fragment
StatusResearch compoundResearch compound
RouteSubcutaneous injectionSubcutaneous injection
Typical vials2 mg, 5 mg, 10 mg2 mg, 5 mg, 10 mg
Dose range250 mcg to 500 mcg2 mg to 5 mg
Frequency1 to 2 times daily1 to 2 times weekly
Half-lifeNo human dataNo human data
Dosing unitmicrogramsmilligrams

Pharmacokinetics

No curves are shown, on purpose.

No published human pharmacokinetics for BPC-157 or TB-500. Drawing one curve against an absent one would imply a comparison that cannot be made.

What the evidence supports

These grade how well each outcome has been studied in humans, never how well either compound works. A strong grade can sit on a negative result.

The strongest evidence for any claimed outcome is Human, observational for TB-500 and Animal only for BPC-157. That gap is the most important line on this page.

BPC-157

OutcomeEvidence
Gastrointestinal protectionAnimal only
Tendon and ligament healingAnimal only
Any outcome in humansNo data

TB-500

OutcomeEvidence
Tissue repair (full-length thymosin beta-4)Human, observational
Tissue repair (TB-500 fragment)Animal only
Any outcome in humans from the fragmentNo data
Human, observationalReported in people without randomization: open-label, single-arm, retrospective, or case series.
Animal onlyShown in animals. No published human trial reports it.
No dataNo published evidence at any level that we could find.

How to choose

Neither has a published human trial

Everything claimed for either comes from rodent work or from reasoning about the parent molecule.

TB-500 is a fragment, and its parent has human data

Full-length thymosin beta-4 has been trialed in people. TB-500 is the actin-binding fragment, and those trials do not transfer to it. See AOD-9604 for how badly that assumption can go.

BPC-157's claim rests on a protein in gastric juice

The 15-residue synthetic sequence is not the same thing as the parent protein doing its job in the stomach.

They are usually combined, so evidence is rarely separated

Community reports almost always describe both at once, which means even the anecdotal signal cannot be attributed to either.

At the syringe

At the default setup on each compound's own page, a 5 mg vial in 2 mL puts 25 mcg on every insulin unit, while a 5 mg vial in 2 mL puts 25 mcg on every unit. That is the number that decides whether a dose lands on a readable mark, and it is set by how much water goes in, not by the compound.

Full references